There is no cure for Herpes viral attacks. Viruses cannot be killed
because they are not alive. Viral attacks, like the common cold, are
acquired from an infected person. The tiny virus, 100,000 times smaller
than a human cell, is a short protein strand similar to a piece of DNA.
Unlike bacteria and germs, viruses are not alive, do not consume food,
and cannot be killed with antibiotics.
Tiny viruses pass through thin cell walls. Cells, unable to differentiate
between DNA and viral protein strands, make billions of copies. These
cells burst, infesting adjacent cells. The immune system alerted by
cell damage, alters the cell walls, making it more difficult for the
virus to enter. Then the body’s immune system floods the area
with fluids to dilute and flush out the virus. The body reacts with
runny nose, congestion, coughing, sneezing, vomiting, diarrhea, etc.
A few viruses enter soft tissues, lying dormant, until a new attack
is triggered. With Herpes viruses, the body flushes out viruses in blisters
and weeping lesions. However, a few Herpes viruses remain, enter nerves,
and are inhibited.
Why do Herpes become dormant in the nerves? Nerves conduct signals.
These signals attract and guide Herpes viruses into the nerves, inhibiting
them. Herpes viruses remain dormant until triggered by nervousness,
sun exposure, fatigue, sickness, etc. initiating a new attack.
Chester A. HEATH, inventor of the Viral Inhibitor, realized, if he
could discover and simulate these nerve signals, he could disrupt and
inhibit viral activities outside the nerves, stopping an attack and
preventing blisters. He did. It is called the BETA signal waveform,
generated by the BETA Viral Inhibitor.
